Dr. Kouri Flores Juan Bautista

ArtsCapítulos y ColaboracionesCurriculum y CongresosDistinciones, Patentes y Tesis


Do chondrocytes undergo "activation" and "transdifferentiation" during the pathogenesis of osteoarthritis? A review of the ultrastructural and immunohistochemical evidence
Prolactin inhibits the apoptosis of chondrocytes induced by serum starvation
Biophysical characteristics of neurotensin polyplex for in vitro and in vivo gene transfection
Ultrastructural and Immunological Characterization of Hepatitis C Core Protein-DNA Plasmid Complexes


Identification of exocytotic membrane proteins, syntaxin-1 and SNAP-25, in Entamoeba histolytica from hamster liver
Expression of MIG-6, WNT-9A, and WNT-7B During Osteoarthritis
Chondrocytes Interconnecting Tracks and Cytoplasmic Projections Observed Within the Superficial Zone of Normal Human Articular Cartilage—A Transmission Electron Microscopy, Atomic Force Microscopy, and Two-Photon Excitation Microscopy Studies


Ultrastructural Evidences of Hepatitis B Infection in Human Liver Biopsies Disclose Complex Assembly and Morphogenesis Pathways for Hepatitis B Virus
Aportes al estudio de los mecanismos patogénicos del VHC y sus implicaciones en la compresión de la enfermedad


Chondrocyte proliferation in a new culture system


Cell death of chondrocytes is a combination between apoptosis and autophagy during the pathogenesis of Osteoarthritis within an experimental model
IRdeseearnchtification of latexin by a proteomic analysis in rat normal articular cartilage